期刊文献+

失神经骨骼肌萎缩与凋亡 被引量:3

暂未订购
导出
摘要 骨骼肌细胞进行性凋亡已成为失神经骨骼肌萎缩机制之一。由于具有特殊的结构和功能,骨骼肌凋亡形态学变化与经典凋亡有所不同,特征之一为骨骼肌细胞核凋亡。各种已知细胞凋亡信号转导途径在骨骼肌细胞凋亡过程中均有发现,但具体调节机制尚存争议。一部分关键调节蛋白的作用较之经典凋亡过程中所起作用有所差异,骨骼肌细胞凋亡与蛋白水解合成的关系有待进一步明确。探索失神经骨骼肌细胞凋亡机制对于维持骨骼肌细胞正常生理功能,阻止失神经骨骼肌萎缩具有十分重要的意义。
作者 潘峰 梁炳生
出处 《国际骨科学杂志》 2010年第3期174-177,共4页 International Journal of Orthopaedics
  • 相关文献

参考文献1

二级参考文献44

  • 1[1]Primeau AJ,Adhihetty PJ,Hood DA.Apoptosis in heart and skeletal muscle.Can J Appl Physiol 2002; 27:349-395
  • 2[2]Earnshaw WC,Martins LM,Kaufmann SH.Mammalian caspases:structure,activation,substrates,and functions during apoptosis.Annu Rev Biochem 1999; 68:383-424
  • 3[3]Cande C,Vahsen N,Garrido C,Kroemer G.Apoptosisinducing factor (AIF):caspase-independent after all.Cell Death Differ 2004; 11:591-595
  • 4[4]Li LY,Luo X,Wang X.Endonuclease G is an apoptotic DNase when released from mitochondria.Nature 2001; 412:95-99
  • 5[5]van Loo G,Schotte P,van Gurp M,Demol H,Hoorelbeke B,Gevaert K,Rodriguez I,Ruiz-Carrillo A,Vandekerckhove J,Declercq W,Beyaert R,Vandenabeele P.Endonuclease G:a mitochondrial protein released in apoptosis and involved in caspase-independent DNA degradation.Cell Death and Differ 2001; 8:1136-1142
  • 6[6]Pollack M,Phaneuf S,Dirks A,Leeuwenburgh C.The role of apoptosis in the normal aging brain,skeletal muscle,and heart.Ann N Y Acad Sci 2002; 959:93-107
  • 7[7]Mayer B,Oberbauer R.Mitochondrial regulation of apoptosis.News Physiol Sci 2003; 18:89-94
  • 8[8]Dupont-Versteegden EE.Apoptosis in muscle atrophy:relevance to sarcopenia.Exp Gerontol 2005; 40:473-481
  • 9[9]Hall PA,Coates PJ,Ansari B,Hopwood D.Regulation of cell number in the mammalian gastrointestinal tract:the importance of apoptosis.J Cell Sci 1994; 107 (Pt 12):3569-3577
  • 10[10]Edelblum KL,Yan F,Yamaoka T,Polk DB.Regulation of apoptosis during homeostasis and disease in the intestinal epithelium.Inflamm Bowel Dis 2006; 12:413-424

共引文献11

同被引文献54

  • 1佟晓杰,张彩顺,曹德寿,刘承吉,于频.脱细胞异体神经移植物桥接大鼠坐骨神经缺损促进神经肌结构重建和功能恢复的实验研究[J].解剖学报,2005,36(1):1-5. 被引量:9
  • 2宋彦红,石国庆,柳楠,代蓉,任航行,刘国庆.肉用绵羊2号染色体GDF8区域4种微卫星标记多态性研究[J].中国畜牧兽医,2007,34(5):54-56. 被引量:11
  • 3漆正堂,卢健.骨骼肌萎缩的细胞分子机制与抗萎缩运动模式[J].中国组织工程研究与临床康复,2007,11(36):7240-7243. 被引量:5
  • 4Wallander HM.Congenital clubfoot.Aspects on epidemiology,residu-al deformity and patient reported outcome[J].Acta Orthop Suppl,2010,339:1-25.
  • 5Dobbs MB,Gumett CA.Update on clubfoot:etiology and treatment[J].Clin Orthop,2009,5:1146-1153.
  • 6Ippolito E,De Maio F,Mancini F,et al.Leg muscle atrophy in idio-pathic congenital clubfoot:is it primitive or acquired[J].J Child Or-thop,2009,3:171-178.
  • 7Flynn JM,Herrera-Soto JA,Ramirez NF,et ai Clubfoot release inmyelodysplasia[J].J Pediatr Orthop,2004,4:259-262.
  • 8Chu A,Labar AS,Sala DA,ei al.Clubfoot classification:correlationwith Ponseti cast treatment[J].J Pediatr Orthop,2010,7:695-699.
  • 9Bouchard C,Dionne FT,Simoneau JA. Genetics of aerobic and anaerobic performances[J].Exercise and Sport Sciences Reviews,1992.27-58.
  • 10VenDITII P,DI Meo S. Antioxidants,tissue damage,and endurance in trained and untrained young male rats[J].Archives of Biochemistry and Biophysics,1996,(01):63-68.

引证文献3

二级引证文献28

相关作者

内容加载中请稍等...

相关机构

内容加载中请稍等...

相关主题

内容加载中请稍等...

浏览历史

内容加载中请稍等...
;
使用帮助 返回顶部