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转化生长因子-β/Smad信号通路及其在肝纤维化中的作用研究进展 被引量:4

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出处 《山西医药杂志(上半月)》 CAS 2009年第1期37-40,共4页 Shanxi Medical Journal
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  • 1Lee T Y, Chin G S, Kim W J, et al. Expression of transforming growth factor beta 1, 2, 3 proteins in keloids D3- Ann Plast Surg, 1999, 43 (2): 179-184.
  • 2Hanasono M M, Kita M, Mikulec A A, et al.Autocrine growth factor production by fetal, keloid,and normal dermal fibroblasts [J]. Arch Facial Plast Surg, 2003, 5 (1):26-30.
  • 3Polo M, Smith, P D, Kim, Y J, et al. Effect of TGF-13 on proliferative scar fibroblast cell kinetics[J]. Ann Plast Surg, 1999,43 (2): 185-190.
  • 4Calderon M, Lawrence W T, Banes A J. Increased proliferation in keloid fibroblasts wounded in vitro [J].J Surg Res, 1996, 61 (2): 343-347.
  • 5Heldin C H, Miyazono K, Dijke P T, TGF-β signalling from cell membrane to nucleus through SMAD protein[J]. Nature, 1997, 390 (6659): 465-471.
  • 6Shaffer J J, Keloidal scars: a review with a critical look at the rapeutic options [J]. J Am Aead Dermntol, 2002(2), 46: S63-97.
  • 7Chin G S, Liu W, Peled Z, et al. Differential expression of transforming growth factor-beta receptors I and Ⅱ and activation of Smad 3 in keloid fibroblasts[J]. Plast Reconstr Surg, 2001, 108 (2) : 423-429.
  • 8Gersdorff G, Susztak K, Rezvani F, et al. Smad3 and Smad4 Mediate Transcriptional Activation of the Human Smad7 Promoter by Transforming Growth Factor [J]. J Biol Chem, 2000, 275 (15):11320-11326.
  • 9Zhou LJ, Oio I, Kaneko F. Role of transforming growth factor-beta 1 in fibroblasts derived from normal and hypertrophic scarred skin. Arch Dermatol Res 1997; 289(11):646-52.
  • 10Shah M, Foreman DM, Ferguson MW. Neutralisation of TGF-beta 1 and TGF-beta 2 or exogenous addition of TGF-beta 3 to cutaneous rat wounds reduces scarring. J Cell Sci 1995; 108(Pt 3): 985-1002.

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