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三氧化二砷对骨髓瘤细胞株RPMI8226作用的研究

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作者 孟建波 赵娟 王金铠 张金巧 康素娴 杨彦 周晨光
机构地区 河北医科大学第三医院血液科 河北医科大学基础医学院细胞生物教研室
出处 《临床荟萃》 CAS 北大核心 2008年第22期1659-1660,共2页 Clinical Focus
基金 河北省科技攻关计划项目(项目编号:062761333)
关键词 多发性骨髓瘤 三氧化二砷 血管内皮生长因子类 基因 BEL-2 细胞凋亡
分类号 R733.302 [医药卫生—肿瘤]
  • 相关文献

参考文献10

  • 1Podar K, Richardson PG, Chauhan D, et al. Targeting the vascular endothelial growth factor pathway in the treatment of multiple myeloma[J]. Expert Rev Anticancer Ther, 2007, 7 (4) :551-566.
  • 2Orpana A, Salven P. Angiogenic and lymphangiogenic molecules in hematological malignancies[J]. Leuk Lymphoma, 2002,43(2) : 219-24.
  • 3孟建波,王金铠,张金巧,康素娴,杨彦.多发性骨髓瘤骨髓单个核细胞分泌VEGF的临床研究[J].天津医药,2008,36(3):222-223. 被引量：1
  • 4孟建波,王金铠,张金巧,王俊祥,常英军.多发性骨髓瘤血管内皮细胞生长因子表达的临床意义[J].临床荟萃,2004,19(15):867-868. 被引量：3
  • 5O'Connor KC, Muhiteh JW, Lacks DJ, et al. Modeling suppression of cell death by Bcl-2 over-expression in myeloma NS0 6A1 cells[J]. Biotechnol Lett,2006,28(23) : 1919-1924.
  • 6Chen Q, Ray S, Hussein MA, et al. Role of Apo2L/TRAIL and Bel-2-family proteins in apoptosis of multiple myeloma[J]. Leuk Lymphoma,2003,44(7) : 1209-1214.
  • 7Bonati A, Rizzoli V, Lunghi P. Arsenic trioxide in hematological malignancies: the new discovery of an ancient drug[J]. Curr Pharm Biotechnol,2006,7(6) :397-405.
  • 8Pettersson HM, Karlsson J, Pietras A, et al. Arsenic trioxide and neuroblastoma eytotoxicity [ J]. J Bioenerg Biomembr, 2007,39(1) : 35-41.
  • 9Gazitt Y,Akay C. Arsenic trioxide: an anti cancer missile with multiple warheads[J]. Hematology, 2005,10(3) : 205-213.
  • 10Engel RH, Evens AM. Oxidative stress and apoptosis: a new treatment paradigm in cancer [J]. Front Biosci, 2006,11 (1) 300-312.

二级参考文献11

  • 1[2]CARMELIET P.Mechanism of angiogenesis and arteriogenesis[J].Nat Med,2000,6(4):389-395.
  • 2[3]de BONT E S,NEEFJES V M E,ROSATI S,et al.New vessel formation and aberrant VEGF/VEGFR signaling in acute leukemia:does it matter? [J].Leuke Lymph,2002,43(10):1901-1909.
  • 3[4]DANKBAR B,PADRO T,LEO R,et al.Vascular endothelial cell growth factor and interleukin-6 in paracrine tumor-stromal cell interaction in multiple myeloma[J].Blood,2000,95(8):2630-2636.
  • 4[5]GUPTA D,TRESON S P,SHIMA Y,et al.Adherence of multiple myeloma cells upragulats vascular endothelial growth factor secretion: therapeutic application[J].Leukemia,2001,15(12): 1950-1961.
  • 5[6]ODAR K,TAI Y,DARIES F E,et al.Vascular endothelial cell growth factor triggers signaling cascades mediating multiple myloma cell growth and migration[J].Blood,2001,98(2):428-435.
  • 6[7]IWASAKI T,HAMANO T,OGATA A,et al.Clinical significance of vascular endothelial growth factor and hepatocyte growth factor in multiple myeloma[J].Br J Haematol,2002,116(4):796-802.
  • 7[8]SEZER O,JAKOB C,EUCKER J,et al.Serum levels of the angiogenic cytokines basic fibroblast growth factor(bFGF),vascular endothelial growth factor (VEGF) and hepatocyte growth factor (HGF) in multiple myeloma[J].Eur J Haematol,2001,66(2):83-88.
  • 8De Bont ES,Neefjes VME,Rosati S,et al. New vessel formation and aberrant VEGF/VEGFR signaling in acute leukemia:Does it matter [J]. Leuke Lymph,2002,43(10) : 1901-1909.
  • 9Tanaka Y, Abe M, Hiasa M, et al. Myeloma cell-osteoclast interaction enhances angiogenesis together with boneresorption: a role for vascular endothelial cell growth factor and osteopontin[J]. Clin Cancer Res,2007,13(3 ) :816-823.
  • 10Mirshahi P,Toprak SK, Faussat AM, et al. Malignant hematopoietic cells induce an increased expression of VEGFR-1 andVEGFR-3 on bone marrow endothelial cells via AKT and roTOR signalling pathways [J]. Biochem Biophys Res Commun, 2006,349 ( 3 ) : 1003- 1010.

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临床荟萃
2008年 第22期

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