3[1]Ascione R, Caputo M, Gomes WJ, et al. Myocardial injury in hypertrophic hearts of patients undergoing aortic valve surgery using cold or warm blood cardioplegia [ J ]. Eur J Cardiothorac Surg,2002,21 ( 3 ) :440 - 446.
4[2]Edwards R, Treasure T, Hosseiu - Nia M, et al. A controlled trial of susbstrate - enhanced, warm reperfusion ( hot shot) versus simperfusion [ J ]. Ann Thorac Surg, 2000,69 ( 2 ): 551 -555.
5[3]Castella M, Buckberg GD,Tan Z. Blood cardioplegic protetion in profoundly damaged hearts: role of Na + - H + exchange inhibition during pretreatment or during controlled reperfusion supplementation[ J]. Ann Thorae Surg,2003,75 (4): 1238 - 1245.
6[4]Gideon C, Michael AB, Richard DW, et al. Intraoperative myocardial protection: current trends and future perspectives [J].Ann Thorac Surg, 1999,68:1995 - 2001.
7De Micheli A, Medrano GA, Iturralde P, et al. Effects of verapamil in ventricular tachycardias: an experimental and clinical study. Acta Cardiol, 1997,52(1):1
8Fauchier L, Babuty D, Poret P, et al. Effect of verapamil on QT interval dynamiciy[J]. Am J Cardiol, 1999,83(5):807.
9Arash Arya,Majid Haghjoo,Zahra Emkanjoo,Amir Farjam Fazelifar,Mohammad Reza Dehghani,Alireza Heydari,Mohammad Ali Sadr-Ameli.Comparison of Presystolic Purkinje and Late Diastolic Potentials for Selection of Ablation Site in Idiopathic Verapamil Sensitive Left Ventricular Tachycardia[J].Journal of Interventional Cardiac Electrophysiology.2004(2)
10Coggins DL,Lee RJ,Sweeney J,Chein WW,Van Hare G,Epstein L,et al.Radiofrequency catheter ablation as a cure for idiopathic tachycardia of both left and right ventricular origin[].Journal of the American College of Cardiology.1994
2Haissaguerre M,Shah DC,Jais P,et al. Role of Purkinjeconductingsys temintriggering of idiopathic ventricularfibrillation. Lancet, 2002,359 : 677.
3Huelsing DJ, Spitzer KW, Pollard AE. Electrotonie suppression of early after depolarizations in isolated rabbit Purkinjemyoeytes. Am J Physiol Heart Circ Physiol,2000,279 : H250.
4Weiss JN,Chen PS, Qu Z, et al. Ventricular Fibrillation: how do we stop the waves from breaking? Circulation Res earch, 2000, 87: 1103.
5Neumar RW, Otto CW, Link MS, et al. Part 8 : adult advanced cardiovascular life support: 2010 American Heart Association Guidelines for Cardiopulmonary Resuscitation and Emergency Cardiovascular Care[J]. Circulation, 2010, 122 ( 18 Suppl 3) : 729- 767.
6Faddy SC,Powell J,Craig JC. Biphasic and monophasic shocks for transthoracic defibrillation: a meta analysis of randomised controlled trials[J]. Resuscitation,2003,58(1) :9-16.
7Link MS, Atkins DL, Passman RS, et al. Part 6: electrical therapies: automated external defibrillators, defibrillation, cardioversion, and pacing: 2010 American Heart Association Guidelines for Cardiopulmonary Resuscitation and Emergency Cardiovascular Care[J]. Circulation, 2010, 122 ( 18 Suppl 3) : S706-719.
8Kaiino K,Iwami T,Berg RA,et al. Comparison of neurological outcomes following witnessed out-of-hospital ventricular fibrillation defibrillated with either biphasic or monophasic automated external defibrillators[J]. Emerg Med J, 2009,26 (7) :492-496.
9Tanabe S, Yasunaga H, Ogawa T, et al. Comparison of outcomes after use of biphasic or monophasic defibrillators among out-of-hospital cardiac arrest patients: a nationwide population-based observational study[J]. Circ Cardiovasc Qual Outcomes, 2012,5 ( 5 ) : 689-696.
10Wang CH, Huang CH, Chang WT, et al. Biphasic versus monophasic defibrillation in out-of-hospital cardiac arrest: a systematic review and meta analysis[J]. Am J Emerg Med, 2013,31(10) : 1472-1478.
