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细胞凋亡与甲状腺疾病

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摘要 细胞凋亡是由基因控制的自主性有序死亡,经特定的程序出现细胞皱缩、染色质浓缩和凋亡小体。细胞凋亡受抑制,会导致自身免疫性疾病或肿瘤,而不恰当的激活则会发生组织器官的退行性病变。
作者 闫朝丽
机构地区 内蒙古医学院第一附属医院内分泌科
出处 《中外医疗》 2008年第13期72-72,共1页 China & Foreign Medical Treatment
关键词 细胞凋亡 甲状腺自身免疫 甲状腺癌
分类号 R581 [医药卫生—内分泌]
  • 相关文献

参考文献9

  • 1段薇,黄华,杜建玲,李昌臣,关宏伟,韩辉.自身免疫性甲状腺疾病甲状腺组织中bcl-2家族蛋白的表达[J].中国地方病学杂志,2005,24(3):271-274. 被引量：4
  • 2史治宙,姬秋和,王瑞华,殷玉华,赵霞,高彬.T_3对人外周血淋巴细胞肿瘤坏死因子相关凋亡诱导配体表达及细胞凋亡的影响[J].中华内分泌代谢杂志,2005,21(5):457-459. 被引量：4
  • 3[3]Chen BK,Ohtsuki Y,Furihata M,et al.Co-overexpression of p53 protein and epidermal growth factor receptor in human papillary treatment carcinomas correlated with lymph node metastasis,tumor size and clinicopathologic stage[J].Int J Oncol,1995,15(5):893～898.
  • 4[4]Pilotti S,Collini P,Rike F,et al.Bcl-2 protein expression in carcinomas originating from the follicular epithelium of the thyroid gland[J].J Pathol,1994,172(4):337～342.
  • 5[5]Hinze R,Gimm O,Taubert H,et al.Regulation of proliferation and apoptosis in sporadic and hereditary medullary thyroid carcinomas and their putative precursor lesions[J].Virchows Arch,2000,437(3):256～263.
  • 6[6]Massart C,Barbet R,Genetet N,et al.Doxorubicin induces Fas-mediated apoptosis in human thyroid carcinoma cells[J].Thyroid,2004,14(4):263～270.
  • 7[7]Nagayama Y,Yokoi H,Takeda K.Adenovirus-mediated tumor suppressor p53 gene therapy for anaplastic thyroid carcinoma in vitro and in vivo[J]:J Clin Endocrinol Metab,2000,85(11):1081～1086.
  • 8[8]Liu Z,Falola J,Zhu X,et al.Antiprolfferative effects of Src inhibition on meduuary thyroid cancerp[J].J Clin Endocrinol Metab,2004,89(7):3503～3509.
  • 9夏燕萍,俞茂华.茶多酚抑制人甲状腺乳头状癌细胞生长[J].中华内分泌代谢杂志,2005,21(4):366-368. 被引量：6

二级参考文献35

  • 1Eguchi K. Apoptosis in autoimmune disease[J]. Intern Med, 2001,40 (4): 275-284.
  • 2Giordano C,Richiusa P, Baguasco M ,et al. Differential regulation of Fas-mediated apoptosis in both thyrocyte and lymphocyte cellular compartments correlates with opposite phenotypic manifestations of autoimmune thyroid disease [J ]. Thyroid, 2001, 11 (3): 233-244.
  • 3Bouillet P,Andreas S. BH3-only proteins-evolutionarily conserved proapoptotic Bcl-2 family members essential for initiating programmed cell death[J]. J Cell Sci, 2002, 11S(Pt8 ): 1567-1574.
  • 4Belinda C,Baliga,Kumar S. Role of Bcl-2 family in maligancy[J].Hematol Oncol, 2002,20 (2): 63-74.
  • 5Shangary S, Johnson DE. Peptides derived from BH3 domains of Bcl-2 family members: a comparative analysis of inhibition of Bcl-2,Bcl-x(L) and Bax oligomerization, induction of cytochrome c release,and activation of cell death[J]. Biochemistry, 2002, 41 ( 30 ): 9485-9495.
  • 6Johnson AL. Editorial: Mcl-1-just another antiapoptotic Bcl-2 homolog[J]? Endocrinology, 1999,140(12) :5465-5468.
  • 7Mitsiades N,Poulaki V,Kotoula V,et al. Fas/Fas ligand up-regulation and Bcl-2 down regulation maybe significant in the pathogenesis of Hashimoto's thyroiditis [J]. J Clin Endocrinol Metab, 1998,83 (6): 2199-2203.
  • 8Judelson C, Privalsky ML. DNA recognition by normal and oncogenic thyroid hormone receptors. Unexpected diversity in half-site specificity controlled by non-zinc-finger determinants. J Biol Chem, 1996,271:10800-10805.
  • 9Gandrillon O, Ferrand N, Michaille JJ, et al. c-erbA alpha/T3R and RARs control commitment of hematopoietic self-renewing progenitor cells to apoptosis or differentiation and are antagonized by the v-erbA oncogene. Oncogene, 1994,9:749-758.
  • 10Brown DD, Wang Z, Kanamori A, et al. Amphibian metamorphosis: a complex program of gene expression changes controlled by the thyroid hormone. Recent Prog Horm Res,1995,50:309-315.

共引文献11

中外医疗
2008年 第13期

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