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极量运动时动脉血乳酸浓度变化与最大氧耗量的相关性 被引量:2

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摘要 极量运动时动脉血乳酸浓度变化与最大氧耗量的相关性钱卫民冯建章孙家珍姚桦伍彩英陈水意人们传统认为乳酸是无氧代谢的产物,体内乳酸堆积是导致机体耐力减低的主要原因[1]。在讨论影响机体做功能力因素时,最大氧耗量和动脉血乳酸浓度是完全对立的两个概念。本试验的...
出处 《中华医学杂志》 CAS CSCD 北大核心 1997年第7期552-553,共2页 National Medical Journal of China
基金 广东省自然科学基金
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参考文献1

  • 1钱卫民,岭南心脏病学杂志,1996年,23卷,22页

同被引文献16

  • 1钱卫民,中华医学杂志,1997年,77卷,552页
  • 2Milavetz JJ, Giebel DW, Christian TF, et al. Time to therapy and salvage in myocardial infarction. J Am Coll Cardiol, 1998,31:1246-1251.
  • 3Christian TF, O'Connor MK, Schwartz RS, et al. Technetium-99m MIBI to assess coronary collateral flow during acute myocardial infarction in two closed-chest animal models. J Nucl Med, 1997,38:1840-1846.
  • 4Pepine CJ, Abrams J, Marks RG, et al. Characteristics of a contemporary population with angina pectoris.TIDES Investigator. Am J cardiol, 1994,74:226-231.
  • 5King LM, Opie LH. Glucose delivery is a major determinant of glucose utilization in the ischemic myocardium with a residual coronary flow. Cardiovasc Res, 1998,39:381-392.
  • 6Young LH, Renfu Y, Russell R, et al. Low-flow ischemia leads to translocation of canine heart GluT-4 and GluT-1 glucose transporters to the sarcolemma in vivo. Circulation, 1997,95:415-422.
  • 7Oliver MF, Opie LH. Effects of glucose and fatty acids on myocardial ischemia and arrhythmias. Lancet, 1994,343:155-158.
  • 8Diaz R, Paolasso EA, Piegas LS, et al. On behalf of the ECLA (Estudios Cardiologics Latinoamerica) collaborative group. Metabolic modulation of acute myocardial infarction. The ECLA glucose-insulin-potassium pilot trial. Circulation, 1998,98:2227-2234.
  • 9Fath-Ordoubadi F, Beatt KJ. Glucose-insulin-potassium therapy for treatment of acute myocardial infarction: an overview of randomized placebo-controlled trials. Circulation, 1997,96:1152-1156.
  • 10Kantor PF, Lucien A, Kozak R, et al. The antianginal drug trimetazidine shifts cardiac energy metabolism from fatty acid oxidation to glucose oxidation by inhibiting mitochondrial long-chain 3-ketoacyl coenzyme A thiolase. Circ Res, 2000,86:580-588.

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