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补体异常与系统性红斑狼疮 被引量：11

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作者 陈竹 汪国生
机构地区 安徽医科大学附属省立医院安徽省立医院风湿科
出处 《中华风湿病学杂志》 CAS CSCD 2005年第9期567-569,共3页 Chinese Journal of Rheumatology
基金 安徽省临床医学重点学科技术进步专项课题(2004Z003)
关键词 补体异常 系统性红斑狼疮 免疫复合物 炎症反应
分类号 R593.241 [医药卫生—内科学]
  • 相关文献

参考文献15

  • 1Man XY, Luo HR, Li XP, et al. Polymerase chain reaction based C4AQ0 and C4BQ0 genotyping: association with systemic lupus erythematosus in southwest han chinese. Ann Rheum Dis, 2003,62: 71-73.
  • 2Mitchell DA, Pickering MC, Warren J, et al. C1q deficiency and autoimmunity: the effects of genetic background on disease expression. J Immunol, 2002, 168: 2538-2543.
  • 3Chen Z, Koralov SB, Kelsoe G. Complement C4 inhibits systemic autoimmunity through a mechanism independent of complement receptors CR1 and CR2. J Exp Med, 2000, 192: 1339-1352.
  • 4Paul E, Pozdnyakova OO, Mitchell E, et al. Anti-DNA autoreactivity in C4-deficient mice. Eur J Immunol, 2002, 32: 2672-2679.
  • 5Nash JT, Taylor PR, Botto M, et al. Immune complex processing in C1q-deficient mice. Clin Exp Immunol, 2001, 123: 196-202.
  • 6Mevorach D, Zhou JL, Song X, et al. Systemic exposure to irradiated apoptotic cells induces autoantibody production. J Exp Med,1998, 188: 387-392.
  • 7Herrmann M, Voll RE, Zoller OM, et al. Impaired phagocytosis of apoptotic cell material by monocyte-derived macrophages from patients with systemic lupus erythematosus. Arthritis Rheum,1998, 41: 1241-1250.
  • 8Nauta AJ, Trouw LA, Daha MR, et al. Direct binding of C1q to apoptotic cells and cell blebs induces complement activation. Eur J Immunol, 2002, 32: 1726-1736.
  • 9Mevorach D, Mascarenhas JO, Gershov D, et al. Complement dependent clearance of apoptotic cells by human macrophages. J Exp Med, 1998, 188: 2313-2320.
  • 10Coremans IE, Spronk PE, Bootsma H, et al. Changes in antibodies to C1q predict renal relapses in systemic lupus erythematosus.Am J Kidney Dis, 1995, 26: 595-601.

二级参考文献8

  • 1Agnello V, Koffler D,Eisenberg JW, et al. C1q in the sera of patients with systemic lupus erythematosus and other hypocoplementemie states: charaterization of high and low molecular weight types. J Exp Med, 1971, 134(Suppl): 228-241.
  • 2Uwatoko S, Mannik M. Low-molecular weight C1q-binding immunoglobulin G in patients with systemic lupus erythematosus consists of autoantibodies to the collagen-like region of C1q. J Clin Invest, 1998, 82: 816-824.
  • 3Horvath L, Czirjak L, Fekete B, et al. High levels of antibodies against C1q are associated with disease activity and nephritis but not with other organ manifestations in SLE patients. Clin Exp Rheumatol, 2001, 19: 667-672.
  • 4Bombardier C, Gladam DD, Urowitz MB, et al. Derivation of the SLEDAI a disease activity index for lupus patients,the committee on prognosis studies in SLE. Arthritis Rheum, 1992, 35: 630-640.
  • 5Monova D, Monov S, Rosenova K, et al. Autoantibodies against C1q: view on association between systemic lupus erythematosus disease manifestation and C1q autoantibodies. Ann Rheum Dis,2002, 61: 563-564.
  • 6Trouw LA, Seelen MA, Duijs JM, et al. Glomerular deposition of C1q and anti-C1q antibodies in mice following injection of antimouse C1q antibodies. Clin Exp Immunol, 2003, 132: 32-39.
  • 7Botto M. C1q knock-out mice for the study of the complement deficiency in autoimmune disease. Exp Clin Immunogenet, 1998,15: 231-234.
  • 8Bernstein KA, Kahl LE, Balow JE, et al. Serological markers of lupus nephritis in patients: use of a tissue-based ELISA and evidence of immunopathogenic heterogeneity. Clin Exp Immunol,1994. 98:60-65.

共引文献7

同被引文献111

引证文献11

二级引证文献79

中华风湿病学杂志
2005年 第9期

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