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糖尿病肾病与IV型胶原的关系 被引量:2

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作者 罗勇
出处 《医学理论与实践》 2005年第7期745-748,共4页 The Journal of Medical Theory and Practice
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参考文献15

  • 1American Diabetes Association. Diabetic nephropathy. Diabetes Care,2003,26(Suppl 1):S94-S98.
  • 2Wtanabe H,stanada H,shigetomi S,et al. Urinary excretion of type IV collagenas a specific in dicator of the progression of diabetic nephropathy.Nephrol,2000,(86):27-35.
  • 3Park IS ,Kiyomoto H,Annoud SL,et al. Expression of transforming growth factor β and IV collagen in early streptozocin induced diabetes.Diabetes,1997,46:924.
  • 4林善锬.糖尿病肾病发病机制的几个关键[J].中华老年多器官疾病杂志,2002,1(3):165-167. 被引量:3
  • 5Okongi H,Nishimura M,Utsunomiya Y,et al. Urinary type IV collagen excretion reflects renal morphological alterations and type IV collagen expression in patients with type 2 diabetes mellitus. Clin Nephrol,2001,55:357-364.
  • 6Cohen MP, Ziyadeh FN, Lautenslager GT,et al. Glycated albumins stimulation of PKC-beta activity is linked to increased collagen IV in measangial cells. Am J Physiol,1999,276:684-690.
  • 7李丹清.胶原代谢与糖尿病肾病[J].国外医学(内分泌学分册),2002,22(3):188-191. 被引量:6
  • 8冯文焕,高鑫,叶真,汪大望,郑景晨,陶志华,吴建波,沈飞霞.尿TGF-β_1、层粘连蛋白、Ⅳ型胶原与2型糖尿病肾病的关系[J].中华内分泌代谢杂志,2003,19(5):393-394. 被引量:5
  • 9Sassy Prigent C,Heudes D,Mandet C, et al. Early glomerular macrophage cruitment in streptozotoicin induced diabetic rats.Diabetes,2000,49:466-475.
  • 10Chen S, Cohen MP,Lautens larger GT, et al. Glycated albumin stimulates TGF beta1 production and prote in kinase C activity in glomeruia rendo the lial cellks.Kidney Int,2001,59:673-681.

二级参考文献25

  • 1林善锬.重视糖尿病肾脏病变的诊断与治疗[J].中华内分泌代谢杂志,1998,14(2):65-67. 被引量:137
  • 2[6]Wolf G, Ziyadeh FN. Molecular mechanisms of diabetic renal hypertrophy. Kidney Int, 1999,56: 393-405.
  • 3[7]Ketteler M, Noble NA, Border WA. Transforming growth factor-β and angiotensin Ⅱ: the missing link from glornerular hyperfiltration to glomerulosclerosis. Annu Rev Physiol,1995,57: 279-295.
  • 4[8]Miller JA. Impact of hyperglycemia on the renin angiotensin system in early human type 1 diabetes mellitus. J Am Soc Nephrol, 1999,10:1778-1785.
  • 5[9]Price DA, Porter LE, Gordon M, et al. The paradox of the low-renin state in diabetic nephropathy. J Am Soc Nephrol,1999,10: 2382-2391.
  • 6[10]Gleizes, PE, Munger JS, Nunes I, et al. TGF-latency: biological significance and mechanisms of activation. Stem Cells, 1997,15:190-197.
  • 7[11]Lane P. Diabetic kidney disease: impact of puberty. Am J Physiol Renal Physiol, 2002,283: F589-F600.
  • 8[12]Yabe-Nishimura C. Aldose reductase in glucose toxicity: a potential target for the prevention of diabetic complications.Pharmacol Rev, 1998,50: 21-33.
  • 9[13]Miller JA, Floras JS, Zinman B, et al. Abnormalities in the renal and vascular responses to LBNP in humans with early diabetes. Am J Physiol, 1994,266: R442-R450.
  • 10Lupia E, Elliot S J, Lenz O.IGF-1 decreases collagen degrad ation in diabetic NOD mesangial cells: implications for diabetic nephropathy[J ].Diabetes,1999,48:1638-1644.

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