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高血压左室肥厚与血金属蛋白酶抑制因子和脑钠素的关系

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出处 《浙江临床医学》 2004年第12期1052-1053,共2页 Zhejiang Clinical Medical Journal
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参考文献7

  • 1[1]Spinale FG,Coker ML, Bond BR,et al. Myocardial matrix degradation and metallopro teinases activation in failing heart: a potential therapeutic target.Cardiovasc Res, 2000,46(2): 225 ~ 238.
  • 2[2]John G, Lainchbury A, Mark Richards M, et al. The effects of pathoph ysiological increments in brain natriuretic peptide in left ventricular sys tolic dysfunction. Hypertension, 1997,30: 398.
  • 3[3]Palatini P, Visentin P, Mormino P, et al. Structural abnormalities and not diastolic dysfuntion are the earliest left ventricular changes in hyperten sion. Am J Hypertens, 1998, (3) 11: 147 ~ 156.
  • 4[4]Diez J,Panizo A,Gil MJ,et al. Serum markers of collagen type Ⅰ metabolism in spontaneously hypertensive rat: relation to myocardial fibrosis.Circilation, 1996,93(5): 1026~ 1032.
  • 5[5]Peterson JT, Li H, Dillon L, et al. Evolution of matrix metalloproteinase and tissue inhibitor expression during heart failure progression in the in farcted rat. Cardiovasc Res,2000,46(2) :307 ~ 315.
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