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肝缺血预处理细胞保护效应中P44/42MAPKs信号通路的作用 被引量：2

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作者 高毅 王瑜 潘明新 孙尔维 单毓强
机构地区 广州第一军医大学附属珠江医院器官移植科
出处 《中华肝胆外科杂志》 CAS CSCD 2004年第3期211-212,共2页 Chinese Journal of Hepatobiliary Surgery
基金 广东省自然科学基金 (No .0 0 10 86)
关键词 肝缺血预处理 细胞保护效应 P44/42MAPKs 信号通路 丝裂原蛋白活化激酶
分类号 R657.3 [医药卫生—外科学]
  • 相关文献

参考文献8

  • 1Fabio M,Maria CA,Marilena F,et al.Extracellual signal-reuglated kinase actvation differentially regulated platelet-derived growth factor's actions in hepatic stellate cells,and is induced by in vivo liver injury in the rat.Hepatology,1999,30:951-958.
  • 2Park KM,kramers C,Vayssier Taussat M,et al.Prevention of kidney ischemia/rperfusion-induced functional injury,MAPK and MAPK kinase activation,and inflammation by remote transient ureteral obstruction.J Biol Chem,2002,277:2040-2049.
  • 3Ping P,Zhang J,Cao X,et al.PKC-dependent activation of p44/p42 MAPKs during myocardial ischemia-reperfusion in conscious rabbits.Am J Physiol,1999,276:H1468-1481.
  • 4Carini R,De Cesaris MG,Splendore R,et al.Signal pathway involved in the development of hypoxic preconditioning in rat hepatocytes.Hepatology,2001,33:131-139.
  • 5Jing Z,Oliver R,Lionel W,et al.The expression of constitutively active isotypes of protein kinase C to investigate preconditioning.J Biol Chem,1998,273:23072-23079.
  • 6Yamashita N,Hoshida S,Nishida M,et al.Time course of tolerance to ischemia-reperfusion injury and induction of heat shock protein 72 by heat stress in the rat heart.J Mol Cell Cardiol,1997,29:1815-1821.
  • 7Bolli R.The late phase of preconditioning.Circ Res,2000,87:972-983.
  • 8Cano E,Mahadevan LC.Parellel signal processing among mammalian MAPKs.Trends Biochem Sci,1995,20:117-129.

同被引文献16

  • 1潘明新,李爱辉,高毅,谢金敏,张翌,张新建,张鸿飞,郭跃虎,张会迎.中国小型猪原位肝移植的实验研究[J].南方医科大学学报,2006,26(7):1069-1070. 被引量：15
  • 2Kume M,Yamamoto Y,Saad S,et al.Ischemic preconditioning of the liver in rats:Implications of heat shock protein induction to increase tolerance ofischemia-reperfusion injury[J].Lab Med,1996,128:2512-22.
  • 3Murry CE,Jennings RB,Reimer KA,et al.Preconditioning with ischemia:a delay of lethal cell injury in ischemic myocardial[J].Circulation,1986,74 (5):1124-36.
  • 4Cave AC.Preconditioning induced protection against post-ischemic contractile dysfunction:characteristics and mechanisms[J].J Mol Cell Cardiol,1995,27 (4):969-79.
  • 5Zacharowski K,Frank S,Otto M,et al.Lipoteichoic acid induces delayed protection in the rat heart:A comparison with endotoxin[J].Arterioscler Thromb Vasc Biol,2000,20(6):1521-8.
  • 6Wu S,Li HY,Wong TM.Cardioprotection of preconditioning by metabolic inhibition in the rat ventricular myocyte involvement of k-opioid receptor[J].Circ Res,1 999,84(12):1388-95.
  • 7Westfall MV,Solaro RJ.Alterations in myofibrillar function and protein profiles after complete global ischemia in rat hearts[J].Circ Res,1992,70(2):302-13.
  • 8Inagaki K,Kihara Y,Hayash ida W,et al.Anti-ischemic effect of a novel cardioprotective agent,JTV519,is mediated through specific activation of delta-isoform of protein kinase C in rat ventricular myocardium[J].Circulation,2000,101 (7):797-804.
  • 9Compagnon P,Lindell S,Ametani MS,et al.Ischemic preconditioning and liver tolerance to warm or cold ischemia:experimental studies in large animals[J].Transplantation,2005,79 (10):1393-400.
  • 10Kume M,Yamamoto Y,Saad S,et al.Is-chemic preconditioning of the liver in rat:Implications of heat shock protein induction to increase tolerance of ischemia-reperfusion injury[J].Lab Med,1996,128:251-252.

引证文献2

二级引证文献4

中华肝胆外科杂志
2004年 第3期

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