A distributed coordinated consensus problem for multiple networked Euler-Lagrange systems is studied.The communication between agents is subject to time delays,unknown parameters and nonlinear inputs,but only with the...A distributed coordinated consensus problem for multiple networked Euler-Lagrange systems is studied.The communication between agents is subject to time delays,unknown parameters and nonlinear inputs,but only with their states available for measurement.When the communication topology of the system is connected,an adaptive control algorithm with selfdelays and uncertainties is suggested to guarantee global full-state synchro-nization that the difference between the agent's positions and ve-locities asymptotically converges to zero.Moreover,the distributed sliding-mode law is given for chaotic systems with nonlinear inputs to compensate for the effects of nonlinearity.Finally,simulation results show the effectiveness of the proposed control algorithm.展开更多
Autapses are synapses that connect a neuron to itself in the nervous system. Previously, both experimental and theoretical studies have demonstrated that autaptic connections in the nervous system have a significant p...Autapses are synapses that connect a neuron to itself in the nervous system. Previously, both experimental and theoretical studies have demonstrated that autaptic connections in the nervous system have a significant physiological function. Autapses in nature provide self-delayed feedback, thus introducing an additional timescale to neuronal activities and causing many dynamic behaviors in neurons. Recently, theoretical studies have revealed that an autapse provides a control option for adjusting the response of a neuron： e.g., an autaptic connection can cause the electrical activities of the Hindmarsh–Rose neuron to switch between quiescent, periodic, and chaotic firing patterns; an autapse can enhance or suppress the mode-locking status of a neuron injected with sinusoidal current; and the firing frequency and interspike interval distributions of the response spike train can also be modified by the autapse. In this paper, we review recent studies that showed how an autapse affects the response of a single neuron.展开更多
An autapse is an unusual synapse that occurs between the axon and the soma of the same neuron. Mathematically, it can be described as a self-delayed feedback loop that is defined by a specific time-delay and the so-ca...An autapse is an unusual synapse that occurs between the axon and the soma of the same neuron. Mathematically, it can be described as a self-delayed feedback loop that is defined by a specific time-delay and the so-called autaptic coupling strength. Recently, the role and function of autapses within the nervous system has been studied extensively. Here, we extend the scope of theoretical research by investigating the effects of an autapse on the transmission of a weak localized pacemaker activity in a scale-free neuronal network. Our results reveal that by mediating the spiking activity of the pacemaker neuron, an autapse increases the propagation of its rhythm across the whole network, if only the autaptic time delay and the autaptic coupling strength are properly adjusted. We show that the autapse-induced enhancement of the transmission of pacemaker activity occurs only when the autaptic time delay is close to an integer multiple of the intrinsic oscillation time of the neurons that form the network. In particular, we demonstrate the emergence of multiple resonances involving the weak signal, the intrinsic oscillations, and the time scale that is dictated by the autapse. Interestingly, we also show that the enhancement of the pacemaker rhythm across the network is the strongest if the degree of the pacemaker neuron is lowest. This is because the dissipation of the localized rhythm is contained to the few directly linked neurons, and only afterwards, through the secondary neurons, it propagates further. If the pacemaker neuron has a high degree, then its rhythm is simply too weak to excite all the neighboring neurons, and propagation therefore fails.展开更多
Synchronization behavior of an ensemble of unidirectionally coupled neurons with a constant input is investigated. Chemical synapses are considered for coupling. Each neuron is also considered to be exposed to a self-...Synchronization behavior of an ensemble of unidirectionally coupled neurons with a constant input is investigated. Chemical synapses are considered for coupling. Each neuron is also considered to be exposed to a self-delayed feedback. The synchronization phenomenon is analyzed by the error dynamics of the response trajectories of the system. The effect of various model parameters e.g. coupling strength, feedback gain and time delay, on synchronization is also investigated and a measure of synchrony is computed in each cases. It is shown that the synchronization is not only achieved by increasing the coupling strength, the system also required to have a suitable feedback gain and time delay for synchrony. Robustness of the parameters for synchrony is verified for larger systems.展开更多
基金supported by the National Natural Sciences Foundation of China(60974146)
文摘A distributed coordinated consensus problem for multiple networked Euler-Lagrange systems is studied.The communication between agents is subject to time delays,unknown parameters and nonlinear inputs,but only with their states available for measurement.When the communication topology of the system is connected,an adaptive control algorithm with selfdelays and uncertainties is suggested to guarantee global full-state synchro-nization that the difference between the agent's positions and ve-locities asymptotically converges to zero.Moreover,the distributed sliding-mode law is given for chaotic systems with nonlinear inputs to compensate for the effects of nonlinearity.Finally,simulation results show the effectiveness of the proposed control algorithm.
基金Project supported by the National Natural Science Foundation of China(Grant Nos.11275084 and 11447027)the Fundamental Research Funds for the Central UniversitiesChina(Grant No.GK201503025)
文摘Autapses are synapses that connect a neuron to itself in the nervous system. Previously, both experimental and theoretical studies have demonstrated that autaptic connections in the nervous system have a significant physiological function. Autapses in nature provide self-delayed feedback, thus introducing an additional timescale to neuronal activities and causing many dynamic behaviors in neurons. Recently, theoretical studies have revealed that an autapse provides a control option for adjusting the response of a neuron： e.g., an autaptic connection can cause the electrical activities of the Hindmarsh–Rose neuron to switch between quiescent, periodic, and chaotic firing patterns; an autapse can enhance or suppress the mode-locking status of a neuron injected with sinusoidal current; and the firing frequency and interspike interval distributions of the response spike train can also be modified by the autapse. In this paper, we review recent studies that showed how an autapse affects the response of a single neuron.
文摘An autapse is an unusual synapse that occurs between the axon and the soma of the same neuron. Mathematically, it can be described as a self-delayed feedback loop that is defined by a specific time-delay and the so-called autaptic coupling strength. Recently, the role and function of autapses within the nervous system has been studied extensively. Here, we extend the scope of theoretical research by investigating the effects of an autapse on the transmission of a weak localized pacemaker activity in a scale-free neuronal network. Our results reveal that by mediating the spiking activity of the pacemaker neuron, an autapse increases the propagation of its rhythm across the whole network, if only the autaptic time delay and the autaptic coupling strength are properly adjusted. We show that the autapse-induced enhancement of the transmission of pacemaker activity occurs only when the autaptic time delay is close to an integer multiple of the intrinsic oscillation time of the neurons that form the network. In particular, we demonstrate the emergence of multiple resonances involving the weak signal, the intrinsic oscillations, and the time scale that is dictated by the autapse. Interestingly, we also show that the enhancement of the pacemaker rhythm across the network is the strongest if the degree of the pacemaker neuron is lowest. This is because the dissipation of the localized rhythm is contained to the few directly linked neurons, and only afterwards, through the secondary neurons, it propagates further. If the pacemaker neuron has a high degree, then its rhythm is simply too weak to excite all the neighboring neurons, and propagation therefore fails.
文摘Synchronization behavior of an ensemble of unidirectionally coupled neurons with a constant input is investigated. Chemical synapses are considered for coupling. Each neuron is also considered to be exposed to a self-delayed feedback. The synchronization phenomenon is analyzed by the error dynamics of the response trajectories of the system. The effect of various model parameters e.g. coupling strength, feedback gain and time delay, on synchronization is also investigated and a measure of synchrony is computed in each cases. It is shown that the synchronization is not only achieved by increasing the coupling strength, the system also required to have a suitable feedback gain and time delay for synchrony. Robustness of the parameters for synchrony is verified for larger systems.
