The explicit expression of the transverse photon wavefunction φγ⊥（u, p2） at the leading twist with the on-shell and the off-shell momenta are calculated in the effective low-energy theory derived from the instant...The explicit expression of the transverse photon wavefunction φγ⊥（u, p2） at the leading twist with the on-shell and the off-shell momenta are calculated in the effective low-energy theory derived from the instanton vacuum of QCD, where both the space-like region as well as the time-like one of the momenta for the virtual photons are investigated. In addition, the problem about the consistency between the two different definitions of the transverse photon wavefunction with chiral-odd Dirac structure, σμν, has been considered, and proven to be true at the leading order by means of the sum rule method. A brief discussion of the dependence of the transverse light-cone photon wavefunction φγ⊥（u, p2） and its coupling, Fγ^⊥（u, P2） to the corresponding quark-antiquark current with respect to p^2, and that about the end-point behaviour of the transverse photon wavefunction is given.展开更多
We present measurements of the γγ＊ → π^0 transition form factor for the momentum transfer range Q^2=4-40 GeV^2 and the γγ＊ → ηc transition form factor for the range Q^2=2-50 GeV^2. The current status of meas...We present measurements of the γγ＊ → π^0 transition form factor for the momentum transfer range Q^2=4-40 GeV^2 and the γγ＊ → ηc transition form factor for the range Q^2=2-50 GeV^2. The current status of measurements of the meson-photon transition form factors for the η and η＇ mesons is discussed. The results of the measurement of the ηc mass, total and two-photon widths are also presented.展开更多
目的探讨m iR-185对胶质瘤细胞增殖、凋亡、自噬、P I3 K/A k t/m T 0 R信号传导通路的影响及其可能存在的作用机制。方法通过构建mimic、inhibitor、mimic+inhibitor胶质瘤细胞U 251以及建立体内移植瘤,应用CCK-8法、FCM法、Western印...目的探讨m iR-185对胶质瘤细胞增殖、凋亡、自噬、P I3 K/A k t/m T 0 R信号传导通路的影响及其可能存在的作用机制。方法通过构建mimic、inhibitor、mimic+inhibitor胶质瘤细胞U 251以及建立体内移植瘤,应用CCK-8法、FCM法、Western印迹与免疫组化技术检测增殖、凋亡、自噬情况。结果m出-185在胶质瘤细胞低表达,™7 M 85高表达可促进细胞凋亡、抑制细胞增殖,增加Caspase-3/9、Beclin-1、LC3II/I、LC3+、P-mTOR、P-AKT、P-PI3K水平,降低Ki67、P62含量,降低移植瘤体积。结论mifl-185可以通过调控细胞增殖、凋亡、自噬相关蛋白水平,促进胶质瘤细胞凋亡与自噬、抑制细胞增殖,可能与抑制PI3K/Akt/m T0R信号通路有关。展开更多
基金Supported by the National Natural Science Foundation of China under Grant No 10075036, the BEPC National Laboratory Project R＆D and BES Collaboration Research Foundation.
文摘The explicit expression of the transverse photon wavefunction φγ⊥（u, p2） at the leading twist with the on-shell and the off-shell momenta are calculated in the effective low-energy theory derived from the instanton vacuum of QCD, where both the space-like region as well as the time-like one of the momenta for the virtual photons are investigated. In addition, the problem about the consistency between the two different definitions of the transverse photon wavefunction with chiral-odd Dirac structure, σμν, has been considered, and proven to be true at the leading order by means of the sum rule method. A brief discussion of the dependence of the transverse light-cone photon wavefunction φγ⊥（u, p2） and its coupling, Fγ^⊥（u, P2） to the corresponding quark-antiquark current with respect to p^2, and that about the end-point behaviour of the transverse photon wavefunction is given.
文摘We present measurements of the γγ＊ → π^0 transition form factor for the momentum transfer range Q^2=4-40 GeV^2 and the γγ＊ → ηc transition form factor for the range Q^2=2-50 GeV^2. The current status of measurements of the meson-photon transition form factors for the η and η＇ mesons is discussed. The results of the measurement of the ηc mass, total and two-photon widths are also presented.
文摘目的探讨m iR-185对胶质瘤细胞增殖、凋亡、自噬、P I3 K/A k t/m T 0 R信号传导通路的影响及其可能存在的作用机制。方法通过构建mimic、inhibitor、mimic+inhibitor胶质瘤细胞U 251以及建立体内移植瘤,应用CCK-8法、FCM法、Western印迹与免疫组化技术检测增殖、凋亡、自噬情况。结果m出-185在胶质瘤细胞低表达,™7 M 85高表达可促进细胞凋亡、抑制细胞增殖,增加Caspase-3/9、Beclin-1、LC3II/I、LC3+、P-mTOR、P-AKT、P-PI3K水平,降低Ki67、P62含量,降低移植瘤体积。结论mifl-185可以通过调控细胞增殖、凋亡、自噬相关蛋白水平,促进胶质瘤细胞凋亡与自噬、抑制细胞增殖,可能与抑制PI3K/Akt/m T0R信号通路有关。
