目的探讨在战创伤早期即采用损伤控制性外科(damage control surgery,DCS)理念并形成临床路径,为改善我军战伤救治进行尝试。方法 2007-2012年收治的按DCS纳入的严重多发伤患者,86例采用整体DCS,56例仍按传统DCS进行救治,对两组的病死...目的探讨在战创伤早期即采用损伤控制性外科(damage control surgery,DCS)理念并形成临床路径,为改善我军战伤救治进行尝试。方法 2007-2012年收治的按DCS纳入的严重多发伤患者,86例采用整体DCS,56例仍按传统DCS进行救治,对两组的病死率及并发症发生率进行对比。结果传统DCS组的病死率及并发症发生率均显著高于整体DCS组(P<0.01)。结论严重多发伤的患者,应尽早积极实施整体DCS,有利于伤员的高效、批量的救治,最大限度地提高战伤救治能力和野战卫勤保障能力。展开更多
Traumatic injury is one of the leading causes of death, with uncontrolled hemorrhage from coagulation dysfunction as one of the main potentially preventable causes of the mortality. Hypothermia, acidosis, and resuscit...Traumatic injury is one of the leading causes of death, with uncontrolled hemorrhage from coagulation dysfunction as one of the main potentially preventable causes of the mortality. Hypothermia, acidosis, and resuscitative hemodilution have been considered as the significant contributors to coagulation manifestations following trauma, known as the lethal triad. Over the past decade, clinical observations showed that coagulopathy may be present as early as hospital admission in some severely injured trauma patients. The hemostatic dysfunction is associated with higher blood transfusion requirements, longer hospital stay, and higher mortality. The recognition of this early coagulopathy has initiated tremendous interest and effort in the trauma community to expand our understanding of the underlying pathophysiology and improve clinical treatments. This review discusses the current knowledge of coagulation complications following trauma.展开更多
文摘目的探讨在战创伤早期即采用损伤控制性外科(damage control surgery,DCS)理念并形成临床路径,为改善我军战伤救治进行尝试。方法 2007-2012年收治的按DCS纳入的严重多发伤患者,86例采用整体DCS,56例仍按传统DCS进行救治,对两组的病死率及并发症发生率进行对比。结果传统DCS组的病死率及并发症发生率均显著高于整体DCS组(P<0.01)。结论严重多发伤的患者,应尽早积极实施整体DCS,有利于伤员的高效、批量的救治,最大限度地提高战伤救治能力和野战卫勤保障能力。
文摘Traumatic injury is one of the leading causes of death, with uncontrolled hemorrhage from coagulation dysfunction as one of the main potentially preventable causes of the mortality. Hypothermia, acidosis, and resuscitative hemodilution have been considered as the significant contributors to coagulation manifestations following trauma, known as the lethal triad. Over the past decade, clinical observations showed that coagulopathy may be present as early as hospital admission in some severely injured trauma patients. The hemostatic dysfunction is associated with higher blood transfusion requirements, longer hospital stay, and higher mortality. The recognition of this early coagulopathy has initiated tremendous interest and effort in the trauma community to expand our understanding of the underlying pathophysiology and improve clinical treatments. This review discusses the current knowledge of coagulation complications following trauma.
