Graduate students universally struggle with vague topics,insufficient innovation,and logical gaps in research proposals,highlighting the need for structured scientific training.This study presents an innovative pedago...Graduate students universally struggle with vague topics,insufficient innovation,and logical gaps in research proposals,highlighting the need for structured scientific training.This study presents an innovative pedagogical model embedding scholarly literature’s logical architecture into LBL-RBL hybrid teaching,implemented in Kunming Medical University’s Neuropathophysiology course.Targeting the complexity of neurological disease mechanisms,the course integrates lecture-based learning(LBL)and research-based learning(RBL)through a small-cohort framework featuring personalized literature-logic embedding→targeted lecture reinforcement→multi-round proposal iteration.Faculty deconstructed domain literature to establish a three-phase training system(“Logic Demonstration-Methodology Mapping-Proposal Embedding”),systematically merging academic logic with research methodology over 9 weeks.Results demonstrate that this problem-driven approach creates authentic scientific inquiry scenarios,activating student knowledge co-construction and collaborative exploration.It successfully enables dynamic competency progression through“cognitive deconstruction→methodological practice→proposal refinement,”significantly enhancing proposal rigor and innovation.This study offers a scalable dual-track solution for cultivating advanced scientific capabilities in medical graduate education.展开更多
Chronic pancreatitis(CP) is a chronic inflammatory disease of the pancreas. The main symptom of patients with CP is chronic and severe abdominal pain. However, the pathophysiology of pain in CP remains obscure.Traditi...Chronic pancreatitis(CP) is a chronic inflammatory disease of the pancreas. The main symptom of patients with CP is chronic and severe abdominal pain. However, the pathophysiology of pain in CP remains obscure.Traditionally, researchers believed that the pain was caused by anatomical changes in pancreatic structure. However, treatment outcomes based on such beliefs are considered unsatisfactory. The emerging explanations of pain in CP are trending toward neurobiological theories. This article aims to review current evidence regarding the neuropathophysiology of pain in CP and its potential implications for the development of new treatments for pain in CP.展开更多
基金The Educational Research Project of Kunming Medical University(2023-JY-Y-024 and 2022-JY-Y-011)Special Research Project for Introduced Talent of Kunming Medical University(J13395001)。
文摘Graduate students universally struggle with vague topics,insufficient innovation,and logical gaps in research proposals,highlighting the need for structured scientific training.This study presents an innovative pedagogical model embedding scholarly literature’s logical architecture into LBL-RBL hybrid teaching,implemented in Kunming Medical University’s Neuropathophysiology course.Targeting the complexity of neurological disease mechanisms,the course integrates lecture-based learning(LBL)and research-based learning(RBL)through a small-cohort framework featuring personalized literature-logic embedding→targeted lecture reinforcement→multi-round proposal iteration.Faculty deconstructed domain literature to establish a three-phase training system(“Logic Demonstration-Methodology Mapping-Proposal Embedding”),systematically merging academic logic with research methodology over 9 weeks.Results demonstrate that this problem-driven approach creates authentic scientific inquiry scenarios,activating student knowledge co-construction and collaborative exploration.It successfully enables dynamic competency progression through“cognitive deconstruction→methodological practice→proposal refinement,”significantly enhancing proposal rigor and innovation.This study offers a scalable dual-track solution for cultivating advanced scientific capabilities in medical graduate education.
文摘Chronic pancreatitis(CP) is a chronic inflammatory disease of the pancreas. The main symptom of patients with CP is chronic and severe abdominal pain. However, the pathophysiology of pain in CP remains obscure.Traditionally, researchers believed that the pain was caused by anatomical changes in pancreatic structure. However, treatment outcomes based on such beliefs are considered unsatisfactory. The emerging explanations of pain in CP are trending toward neurobiological theories. This article aims to review current evidence regarding the neuropathophysiology of pain in CP and its potential implications for the development of new treatments for pain in CP.
