The bronze acupuncture model was produced by Emperor Qianlong’s order in 1744 A.D.It has been 274 years since then,and this model has always been well kept and handed down with a full record.It is of great traditiona...The bronze acupuncture model was produced by Emperor Qianlong’s order in 1744 A.D.It has been 274 years since then,and this model has always been well kept and handed down with a full record.It is of great traditional medical and cultural value,regarded as the treasure of Shanghai Museum of Traditional Chinese Medicine due to its rareness and intactness both at home and abroad.展开更多
Over the last three decades,microglia have been recognized as essential components of central nervous system(CNS)development,homeostasis,immune surveillance,and neurodegenerative pathogenesis(Lin&Wang,2024;Prinz e...Over the last three decades,microglia have been recognized as essential components of central nervous system(CNS)development,homeostasis,immune surveillance,and neurodegenerative pathogenesis(Lin&Wang,2024;Prinz et al.,2019).Historically,microglia were regarded as exclusive to the CNS,based on the absence of cells bearing microglial morphology,transcriptional identity,and ontogeny in tissues outside the CNS in standard mouse and rat models.展开更多
文摘The bronze acupuncture model was produced by Emperor Qianlong’s order in 1744 A.D.It has been 274 years since then,and this model has always been well kept and handed down with a full record.It is of great traditional medical and cultural value,regarded as the treasure of Shanghai Museum of Traditional Chinese Medicine due to its rareness and intactness both at home and abroad.
基金supported by the National Natural Science Foundation of China(32425024)Shenzhen Medical Research Fund(B2302006)。
文摘Over the last three decades,microglia have been recognized as essential components of central nervous system(CNS)development,homeostasis,immune surveillance,and neurodegenerative pathogenesis(Lin&Wang,2024;Prinz et al.,2019).Historically,microglia were regarded as exclusive to the CNS,based on the absence of cells bearing microglial morphology,transcriptional identity,and ontogeny in tissues outside the CNS in standard mouse and rat models.
