This paper gives a practical schema for using DSP boards to construct Vehicle License Plate Recognition (VLPR) modules that could be embedded in any Intelligent Transportation System (ITS). Using DSP can avoid the hea...This paper gives a practical schema for using DSP boards to construct Vehicle License Plate Recognition (VLPR) modules that could be embedded in any Intelligent Transportation System (ITS). Using DSP can avoid the heavy investment in dedicated VLPR system and improve the computational power compared to PC software environment. Low cost, high computational power, and high flexibility of DSP provide the License Plate Recognition System (LPRS) an excellent cost-effective solution to execute the major part of the recognition tasks. This paper describes a successful implementation of VLPR system based on Texas Instruments (TI)'s TMS320DM642. The DSP board acquires video (which could be output to a monitor for surveillance) from a camera, captures images from the video, locates and recognizes the license plates in images, and then sends the recognized results and related images after compression to a host PC through the network. Finally, the overall software is optimized according to the features of DM642 chip. Experiments showed that the DSP VLPR system performs well on the local license plates, and that the processing speed and accuracy can meet the requirement of practical applications.展开更多
目的基于MyD88/NF-κB通路探讨芪黄健脾滋肾颗粒(QJZ)改善MRL/lpr小鼠肾损害的作用机制。方法6只雌性C57BL/6小鼠为正常对照组(Control组),将30只雌性MRL/lpr小鼠随机分为模型组(Model组)、芪黄健脾滋肾颗粒低剂量组(QJZ-L组)、芪黄健...目的基于MyD88/NF-κB通路探讨芪黄健脾滋肾颗粒(QJZ)改善MRL/lpr小鼠肾损害的作用机制。方法6只雌性C57BL/6小鼠为正常对照组(Control组),将30只雌性MRL/lpr小鼠随机分为模型组(Model组)、芪黄健脾滋肾颗粒低剂量组(QJZ-L组)、芪黄健脾滋肾颗粒中剂量组(QJZ-M组)、芪黄健脾滋肾颗粒高剂量组(QJZ-H组)、泼尼松组(Pred组),6只/组。干预8周后取各组小鼠的尿液、全血及肾脏组织,生化试剂盒检测肾损害指标:24 h尿蛋白定量(24 h PRO)、尿总蛋白肌酐比值(UTPCR)、尿蛋白肌酐比(UACR)。ELISA法检测血清中免疫球蛋白G(IgG)、补体3(C3)、补体4(C4)、抗双链DNA抗体(antidsDNA)、干扰素γ(IFN-γ)、白细胞介素17(IL-17)。肾脏组织进行HE染色和透射电镜观察肾小球超微结构。采用RT-qPCR、Western blotting和免疫组化法检测肾组织中MyD88/NF-κB通路相关分子的表达。结果与Model组相比,QJZ-L、QJZ-M、QJZ-H和Pred组24 h PRO、UTPCR、UACR、IgG、anti-dsDNA、IFN-γ、IL-17降低(P<0.01),C3、C4水平升高(P<0.01)。HE染色结果显示,与Model组比较,各组肾小球内皮细胞增生、系膜增厚减轻。透射电镜显示,各组肾小球电子致密物沉积、炎性细胞浸润较Model组减轻。RT-qPCR和免疫组化结果显示,与Model组相比,QJZ-L、QJZ-M、QJZ-H和Pred组肾脏中MyD88、NF-κB表达下降(P<0.05)。Western blotting结果显示,各组肾脏中p65、p52蛋白水平低于Model组(P<0.01)。结论芪黄健脾滋肾颗粒能够改善MRL/lpr小鼠肾损害,其机制可能与抑制MyD88/NF-κB通路过度激活有关。展开更多
基金the National Natural Science Foundation of China (No. 60473106)the Hi-Tech Research and Development Program (863) of China (Nos. 2007AA01Z311 and 2007AA04ZA5)
文摘This paper gives a practical schema for using DSP boards to construct Vehicle License Plate Recognition (VLPR) modules that could be embedded in any Intelligent Transportation System (ITS). Using DSP can avoid the heavy investment in dedicated VLPR system and improve the computational power compared to PC software environment. Low cost, high computational power, and high flexibility of DSP provide the License Plate Recognition System (LPRS) an excellent cost-effective solution to execute the major part of the recognition tasks. This paper describes a successful implementation of VLPR system based on Texas Instruments (TI)'s TMS320DM642. The DSP board acquires video (which could be output to a monitor for surveillance) from a camera, captures images from the video, locates and recognizes the license plates in images, and then sends the recognized results and related images after compression to a host PC through the network. Finally, the overall software is optimized according to the features of DM642 chip. Experiments showed that the DSP VLPR system performs well on the local license plates, and that the processing speed and accuracy can meet the requirement of practical applications.
文摘目的基于MyD88/NF-κB通路探讨芪黄健脾滋肾颗粒(QJZ)改善MRL/lpr小鼠肾损害的作用机制。方法6只雌性C57BL/6小鼠为正常对照组(Control组),将30只雌性MRL/lpr小鼠随机分为模型组(Model组)、芪黄健脾滋肾颗粒低剂量组(QJZ-L组)、芪黄健脾滋肾颗粒中剂量组(QJZ-M组)、芪黄健脾滋肾颗粒高剂量组(QJZ-H组)、泼尼松组(Pred组),6只/组。干预8周后取各组小鼠的尿液、全血及肾脏组织,生化试剂盒检测肾损害指标:24 h尿蛋白定量(24 h PRO)、尿总蛋白肌酐比值(UTPCR)、尿蛋白肌酐比(UACR)。ELISA法检测血清中免疫球蛋白G(IgG)、补体3(C3)、补体4(C4)、抗双链DNA抗体(antidsDNA)、干扰素γ(IFN-γ)、白细胞介素17(IL-17)。肾脏组织进行HE染色和透射电镜观察肾小球超微结构。采用RT-qPCR、Western blotting和免疫组化法检测肾组织中MyD88/NF-κB通路相关分子的表达。结果与Model组相比,QJZ-L、QJZ-M、QJZ-H和Pred组24 h PRO、UTPCR、UACR、IgG、anti-dsDNA、IFN-γ、IL-17降低(P<0.01),C3、C4水平升高(P<0.01)。HE染色结果显示,与Model组比较,各组肾小球内皮细胞增生、系膜增厚减轻。透射电镜显示,各组肾小球电子致密物沉积、炎性细胞浸润较Model组减轻。RT-qPCR和免疫组化结果显示,与Model组相比,QJZ-L、QJZ-M、QJZ-H和Pred组肾脏中MyD88、NF-κB表达下降(P<0.05)。Western blotting结果显示,各组肾脏中p65、p52蛋白水平低于Model组(P<0.01)。结论芪黄健脾滋肾颗粒能够改善MRL/lpr小鼠肾损害,其机制可能与抑制MyD88/NF-κB通路过度激活有关。
