1[1]Mullenheim J, Ebel D, Frassdorf J, et al. Isoflurane preconditions myocardium against infarction via release of free radicals[J]. Anesthesiology, 2002, 96(4):934-940.
2[2]Tanaka K, Weihrauch D, Kehl F,et al. Mechanism of preconditioning by isoflurane in rabbits: a direct role for reactive oxygen species[J] . Anesthesiology, 2002 ,97(6):1485-1490.
3[3]Novalija E, Varadarajan SG, Camara AK,et al. Anesthetic preconditioning: Triggering role of reactive oxygen and nitrogen species in isolated hearts[J] . Am J Physiol Heart Circ Physiol, 2002, 283(1): H44-52.
4[4]Pain T, Yang XM, Critz SD, et al. Opening of mitochondrial KATPchannels triggers the preconditioned state by generating free radicals[J]. Circ Res, 2000, 87(6):460-466.
5[5]Kevin LG, Novalija E, Riess ML, et al. Sevoflurane exposure generates superoxide but leads to decreased superoxide during ischemia and reperfusion in isolated hearts[J]. Anesth Analg, 2003, 96(4):949-955.
6[6]Hanley PJ, Ray J, Brandt U,et al. Halothane, isoflurane and sevoflurane inhibit NADH:ubiquinone oxidoreductase (complex I) of cardiac mitochondria[J]. J Physiol, 2002, 544 (Pt 3): 687-693.
7[7]Ludwig LM, Tanaka K, Eells JT,et al. Preconditioning by isoflurane is mediated by reactive oxygen species generated from mitochondrial electron transport chain complex III[J]. Anesth Analg, 2004, 99(5):1308-1315.
8[8]Riess ML, Eells JT, Kevin LG, et al. Attenuation of mitochondrial respiration by sevoflurane in isolated cardiac mitochondria is mediated in part by reactive oxygen species[J]. Anesthesiology, 2004 , 100(3):498-505.
9[9]Riess ML, Kevin LG, McCormick J, et al. Anesthetic preconditioning: the role of free radicals in sevoflurane-induced attenuation of mitochondrial electron transport in Guinea pig isolated hearts[J]. Anesth Analg, 2005, 100(1):46-53.
10[10]Zhang DX, Chen YF, Campbell WB, et al. Characteristics and superoxide-induced activation of reconstituted myocardial mitochondrial ATP-sensitive potassium channels[J]. Circ Res, 2001, 89(12): 1177-1183.